Who Speaks for the Unborn Brains?


 

Majid Ali, M.D.

140 West End Avenue,   Suite 1-H, New York, NY 10023

212-873-2444 344

344 Prospect Avenue, Suite 3-C, Hackensack, NJ. 07610

201-996-0027

Askipm@aol.com


 

                            TM
The GIO Path

The GIO Path of Health and Healing


 

The Core Goals of The GIO Path of Nurturing the Unborn Brain With Philosophy and Sceince of Health and Healing

For Parents and expecting parents Willing and Able to Learn Valuable Information About Their Children:

1. To learn and understand the uneven medical playing fields which turn corporate medical America into sellers and patients into buyers

(In Medical Seller-Buyer Dealings, the Short-Changed Party Is Always the Buyer.) 

2. Level their medical playing fields in countering corporate medical America;

3. To study and learn well the philosophy and science of holism in health and healing;

4. To effectively counter corporate deceptions as well as idealogical distortions of ill-informed health activists;

5. To study and learn well the philosophy and science of holism in health and healing;

6. To undertake, for love of learning, a  path of self-learning to self-empowerment to self-healing

5. Through personal observations learn how the sound science of holism in health and healing To as Become their own well-informed personal health advocates;

6. To learn to preserve health, prevent disease, and reverse chronic diseases by becoming their own primary physicians;

7. To remain students of the above subjects and yet serve as other as teachers in the  field.

******

GIO Path for Expecting Mothers and fathers

GIO Path is Healing Is a Path of Self-Learning to Self-Empowering to Self- Healing with a Sharp Focus on

1. Self-Education

2. Self-Empowerment

3. Health Preservation

4. Disease Prevention, and

5. Reversing chronic disease.


I must be kind to my body so my body can be kind to me.


Ali GIO Health and Healing Course is a free public service health course designed for patient education and patient empowerment.

Ali GIO Health and Healing Course is suitable for students of all descriptions – from elementary school to doctorate students, from patients to physicians, from public health policy makers to philosophers of health, hope, and healing.


 

Seven Parts of Dr. Ali GIO Health and Healing Course

  1. GIO Path Health and Healing Course Introduction – Three Root Problems, Three Sets of Solutions

  2. GIO Path GIO Health and Healing Course, Part One – Self-learning and Self-Empowering for Self-Healing Chronic Disease

  3. GIO Path Health and Healing Course, Part Two –Strong Scientific Claims Require Strong Scientific Evidence

  4. GIO Path Health and Healing Course, Part Three – Energy Thinking for Energy Healing

  5. GIO Path Health and Healing Course, Part Four – Safety First, Acute and Chronic Diseases

  6. GIO Path Health and Healing Course, Part Five – Top Seven for Gut, Oxygen, and Insulin

  7. GIO Path Health and Healing Course, Part Six – The Gut-Liver-Brain Connections

  8. GIO Path Health Healing Course, Part Seven – The Spiritual Dimension and Course Study Materials


Why Do I Focus on Oxygen, Gut, and Insulin in All Matters of Health and Healing?

 

My Simple Answer: Because in my sixty-one year study, research, teaching, publications, and clinical practice,  I observed the best long-lasting results in all chronic diseases (including cancer) with my GIO philosophy and science, the three essential areas are simply stated below:

  1.       for Gut ecology and gut microbiome
  2.        for Insulin homeostasis
  3.        for molecular Biology of oxygen

To introduce readers to my academic background and qualifications to address all matters of health and healing:

  1. I am a surgeon (FRCS, Eng)-turned – pathologist (Columbia University, New York) -turned – nutritionist – turned clinical ecologist – turned integrationist;
  2. I am the solo author of all 12 volumes of The Principles and Practice of medicine  (1989-2009), and 27 other books (see my CV on this site for details).
  3. I published 215 papers (many with my research and clinical collaborators) in eminent medical journals (including Lancet, JAMA, Cancer, Kidney International, Am Heart Journal, Am J Clinical pathology, Archives of Pathology, Israeli Journal of Sciences, Environmental Medicine, and others). See my CV on this site for details).
  4. My CV includes a large number of publications on three subjects of molecular biology of oxygen, gut ecology and gut microbiome, and insulin homeostasis.

 


 

The Autism-Inflammation-Infection Links

The Oxygen Model of the Autism Spectrum predicts that pathologic (disease-causing) inflammation from any source will worsen the signs and symptoms associated with spectrum.

My Oxygen Model of Autism is a unifying model that explains all aspects of autism on the basis of disruptions of oxygen homeostasis occurring during fetal life, infancy, and childhood. This model covers causes, clinical course, consequences, and control of clinical problems associated with the autism spectrum. It is based on a biologist’s holistic view of the brain development and neurotransmission. The autism spectrum is not a range of psychological or psychiatric disorders.

Inflammation of both healing and disease-causing types are governed by oxygen. The Oxygen Model of the Autism Spectrum predicts that pathologic (disease-causing) inflammation from any source will worsen the signs and symptoms associated with spectrum. This has been amply borne by my clinical experience. Parents of children on the spectrum under my care regularly recognize this connection regularly. The crucial clinical significance of this statement is: Every efforts should be made to detect and effectively address all sources of inflammation in all individuals on the spectrum, whether toddlers, children, teenagers, or adults.

Inflammation During Fetal Life
The Oxygen Model of Autism also predicts that exposure to the effects of maternal pathologic inflammation from any source will increase the risk of the unborn baby developing autism later in life. This was documented clearly by a study published online May 5 in The Journal of Autism and Developmental Disorders which fully validated my clinical observations with its study. Specifically, children of women who had a febrile illness during pregnancy were more than twice as likely as those who did not to develop autism and related developmental disorders. The study included 701 children with autism spectrum disorders or developmental delays and 421 normal controls.

In past reports, some data pointed to a connection between autism and various infections during pregnancy, including measles, mumps, rubella and influenza.


 

Unhealthy Womb Conditions for the Unborn Brain

For more than three decades I have strived to raise awareness of the toxic conditions of the womb to which the unborn are exposed to incrementally during the time of their greatest vulnerability, the first three months of gestation. The title of this article in my Child Development Course should not surprise anyone. Toxicities of the womb do not begin with the pregnancy. Rather they accumulate steadily if the future mother’s:

* Functional nutritional deficiencies

* Undetected and unmanaged food, mold, and other allergies

* Sugar and antibiotic abuse

* Stress of frustrations, disappointments, resentment, and anger

* Substance abuses

.* Environmental pollutants

* Toxic metal load


 

The Oxygen Model of Mental Health and Disorders

I proposed my Oxygen Model of Mental Health and Disorder as an extension of my Oxygen Model of Health and Disease. It is a unifying model that explains all aspects of mental health and mental disorders—causes, clinical course, consequences, and control—on the basis of altered oxygen signaling. These abnormalities essentially begin during fetal life and continue through infancy, and childhood. This model is based on a biologist view of the brain development and neurotransmission. Autism is not a psychological or psychiatric disorder.

* Articles in Child Development Course

 

 



The mission of Children’s Health Corps is to offer parents, teachers, and other interested parties information about health preservation and disease prevention that is free of ideological distortions and corporate deceptions.

 

 

 

 

The educational purpose of Children’s Health Corps sharply focuses on the subjects of:

  1. Nutrition
  2. Environmental toxins
  3. Stress
  4. Natural immunity
  5. Specific threats to the immune syastem.

This site is based on the works of Majid Ali, MD whose over 50 years in medicine has given him a unique perspective in the evolution of children’s health issues that will effect the 21st century.


 

Human Canaries on the Autism Spectrum

Majid Ali, M.D.

Children and others on the autism spectrum make a clarion call for the future of humankind. They teach us that humans have a finite capacity to sustain and survive existential threats to their brain cells. Specifically, they inform that environmental toxicities that put in jeopardy the oxygen order and energetics of their brain cells can be expected to affect all humans with time. Simply stated, they are human canaries on the autism spectrum.

Fatigue Canaries

I opened my book The Canary and Chronic Fatigue Syndrome (1994) with the following words: “Chronic fatigue sufferers are human canaries—people who tolerate poorly the biologic stressors of the late 20th cenutury…[They] are telling us something important about the shape of things to come. None of us are really immune to oxidative stressors that fatigue them.” My point then: Given planetary chemicalization and “angerization” trends, Nearly all humans in the future can be expected to develop fatigue that is considered undue today. How could I have imagined that in merely two decades I would hear the same clarion call from children with autism and related disorders on the spectrum.

Autism Canaries, One in 29 Boys

Consider the following quote from the November 6, 2012 issue of New York magazine: “Before 1980, one in 2,000 children was thought to be autistic. By 2007, the Centers for Disease Control were reporting that one in 152 American children had an autism-spectrum disorder. Two years later, the CDC updated the ratio to one in 110. This past March, the CDC revised the number upward again, to one in 88 (one in 54, if you just count boys, who are five times as likely to have one as girls). A South Korean study from last year put the number even higher, at one in 38. And in New Jersey, according to the latest numbers, an improbable one in 29 boys is on the spectrum.”

Autism Is Not a Psychologic Disorder

Nor is it a psychiatric disorder. To think so, as I have shown in articles on my Oxygen Modebelow to order l of Autism, is a grave injustice to children and others on the autism spectrum. Psychologists and psychiatrists do not study nor practice nutritional medicine. Nor do they study or practice environmental medicine. Stress simply cannot be managed without effectively addressing nutritional and environmental issues relevant to individuals on the spectrum. I discuss this subject at length in two video seminars linked below. Please click on the boxes to order them

Our planet is warming and so are we. Our planet is acidifying and so are we. Our planet is fermenting and so are we. Our planet is chemicalized, overpopulated, and angerized.  All these factors are seriously affecting animals and plants. We cannot be immune these adverse effects. In this article, I reproduce from The New York times an extraordinary group of photographs and accompanying texts. In other articles in this series, I offer additional comments on these important subjects.

Evolution Under Our Noses

This is evolution under our noses. This, of course, is not new. What is new is the scale of the change. Humans can think only on linear scales. For example, I can say that going at 50 miles an hour, my car will travel 100 miles in two hours. This is clear cut math. That is not the way of nature however. Natures moves on an exponential scale. The changes are not clear cut because nature moves within its own parameters with steps and counter-steps, masking the rate of change. There are of cours exceptions to it on geologic scales when violent volcanic or asteroid activity can change thing smore quickly on th eevolutionary scale.

Climatic Changes of Chaos Scale

The evidence is rapidly accumulating that the current climatic changes are likely to build up changes on exponential scale that may impact us the same way the catastrophic extinction events in our past did. A rapid acceleration of evolutionary progress down a completely different path than nature intended.

Consider the case of the rising prevalence of autism spectrum, I rest my case by reproducing here a dramatic graph reproduced from the science journal, Nature (Nov.3, 2011):


What impacts the tea harvest – impacts Autism

In China, the tea harvest depends on the monsoons: The best tea is harvested in springtime, when the weather is still dry. But climate change threatens to extend the monsoon season.

“Post monsoon season, farmers get much less from their harvest, and a lot of the chemicals that give tea its flavor drop,” said Colin M. Orians, a chemical ecologist at Tufts University. “If climate changes the onset of the monsoon season, farmers will have a shorter window in which to harvest their tea.”

Over the next four years, Dr, Orians and his team will investigate the effects of changing temperatures and rainfall on tea quality and on the livelihoods of farmers who depend on the harvest.

Weather changes that effect tea – effect people


Shrinking Salamanders

Salamanders in the Appalachian Mountains are getting smaller, and species at lower altitudes, where the greatest drying and warming has occurred, are the most affected. One species became 18 percent smaller over 55 years.
t-weight:400;”>“It could be that a change in body size is the first response to climate change,” said Karen Lips, an ecologist at the University of Maryland. “Their food may be affected, and they may be producing smaller babies.”

 

Dr. Lips partially relied on the data of Richard Highton, a retired ecologist from the University of Maryland who spent 50 years studying and collecting salamanders that are now preserved at the Smithsonian Institution. At the time of his retirement, he noted that salamanders were mysteriously disappearing.

“If they are not nearly as big, they may not be producing as many offspring,” Dr. Lips said.

To test the theory, Dr. Lips and her team plan to raise salamanders in incubators that mimic different climates.


The Bumble Bee Die Out

SINDYA N. BHANOO

Bumblebees and other pollinators are critical to global agriculture, but recent studies suggest that up to one-quarter of Europe’s bumblebee population may die out.

Researchers say that climate change is at least partly to blame, along with disease and loss of habitat.

Scientists estimate that pollinators indirectly contribute about $30 billion a year to the European economy. “Pollinators are essential to our population,” said Jean-Christophe Vié, deputy director of the species program at the International Union for Conservation of Nature in Switzerland.


Roe Deer

Roe deer, a small, reddish-brown species that flourishes all over Europe, give birth when new plant growth provides ample nutritious food for the mother. But flowers are blooming earlier than they used to, and the deer are missing their meals.Researchers tracked deer births from 1985 to 2011 in the Champagne region of northeastern France, where average spring temperatures have steadily increased and flowering time is coming gradually earlier. The study is online in PLOS Biology.

The deer time their fertility by light availability, not temperature. With earlier springs, they are now giving birth too late to take advantage of the best food.

Using data on 1,095 births, the scientists calculated that the mismatch between flowering time and birth over the period had grown by 36 days.

The researchers estimate that deer fitness declined by 6 percent over the period, and by 14 percent in 2007 and 2011, when flowering was particularly early.

Roe deer are very dependent on large quantities of high quality food, and the critical stage is the first week’s supply,” said the lead author, Jean-Michel Gaillard, a director of research at the National Center for Scientific Research at the University of Lyon. “Unlike birds, for example, that can migrate and breed earlier, roe deer cannot.”


The Olive Tree Crisis

NICHOLAS BAKALAR
In the Mediterranean Basin, small olive farms can support entire families. Olive trees are notoriously drought-resistant, and even in arid ecosystems they attract migratory birds and a host of insect species. But as the region warms, some olive trees will not be as productive.

“In the south, you’re going to see a lower crop yield,” said Andrew Paul Gutierrez, an ecologist at the University of California, Berkeley. “In marginal areas, the farmers will just go out of business.”

Dr. Gutierrez and his colleagues predict that some local farmers ultimately will have to abandon their orchards, leaving barren swaths of desert where biodiversity once flourished.


The Lemming Natural Causes Die Off

JOSHUA A. KRISCH

Contrary to myth, lemmings do not commit mass suicide. But populations do rise and fall in predictable cycles, to the benefit and detriment of predators like arctic foxes and migratory birds.

Recently, scientists noticed that some groups of lemmings have died off.

“The lemming cycle is the heartbeat of the terrestrial Arctic,” said Nicolas Lecomte, a biologist at the University of Moncton in Canada. “Now we’re seeing the collapse of the main prey of many terrestrial predators.”

Lemmings survive harsh winters by hiding in the snow. When warmer temperatures bring off-season rain, that snow turns to ice, and the lemmings cannot burrow.

Dr. Lecomte has found that as lemmings die off en masse, the fragile Arctic ecosystem is growing weaker.

Wheat, Rice and Corn

If wheat, rice and corn are going to continue to feed the world, the crops will have to adapt to warmer temperatures. The latest report from the Intergovernmental Panel on Climate Change offers some predictions. published last spring in Nature Climate Change, concluded that a 3.6 degree Fahrenheit increase in temperature will bring a significant decline in crop yields.

Most projections see a decrease from 2030 onward, with greater decreases in the 2040s and 2050s.

Selective breeding and changes in irrigation methods, pest control, fertilization and planting dates may compensate, partially, for the temperature change. But most of these adaptations will work better in temperate regions, while tropical crop yields will continue to decline.

Extreme weather events — another consequence of climate change — will affect yields year-to-year in ways that are difficult to forecast.

“There are two pieces of bad news here,” said the lead author of the I.P.C.C report, Andy J. Challinor, a professor of climate impact at the University of Leeds in England. “One is that average yields are going down. The other is that yields in any given year will be less reliable.”


Sharks who can’t smell blood

NICHOLAS BAKALAR

Sharks pursue their prey partly by odor, but rising carbon dioxide levels may severely impair their sense of smell.  Scientists used the smooth dogfish, a small shark, as an experimental animal. They created tanks in which some jets of water held the odor of squid, a favorite food, or no odor at all. The water in the tanks also contained varying levels of carbon dioxide.

With carbon dioxide levels resembling today’s, the sharks spent 60 percent of their time nosing about the plume with the squid odor. But in water with carbon dioxide concentrations predicted for the year 2100, the animals actively avoided the jet with the food odor, spending only 15 percent of their time there.

Any change in shark feeding habits might affect other species as well.

“There might be a decrease in hunting behavior among sharks, and an increase in prey animals as a result,” said a co-author, Ashley R. Jennings, a researcher at Boston University. “That’s assuming the prey animals aren’t being affected by CO₂ as well.”


Snails that don’t jump

NICHOLAS BAKALAR

Conch Snails

As the oceans gather carbon, a small sea snail that lives in the Great Barrier Reef risks losing its famous ability to leap.t-weight:400;”The conch snail jumps to escape from a predator, also a sea snail, that tries to inject it with a poisonous dart.

In laboratory experiments in water with increased carbon dioxide levels, the snails were 50 percent less likely to jump. And snails that did jump took nearly twice as long to do so.

The carbon dioxide and acidity disrupt a neurotransmitter receptor in the snail’s nervous system, one that other marine animals also rely on.

“They are very widespread,” said Sue-Ann Watson, a biologist at James Cook University in Australia. “It could affect many marine animals and their behaviors.”

Oceans today are 30 percent more acidic than they were 250 years ago, when the Industrial Revolution started. And it is getting worse.

“By the end of the century, if we carry on with business as usual, they will be 150 percent more acidic than they were 250 years ago,” Dr. Watson said.


Moss adapts and proves humans cause climatic changes

SINDYA N. BHANOO

As Arctic temperatures rise every summer, some of the ice on Canada’s Baffin Island melts, revealing the moss trapped underneath. Now, using radiocarbon dating, researchers have determined that until recently some of that moss hadn’t seen daylight in 44,000 years. The melting ice not only gives scientists the chance to study ancient moss, but adds to evidence that climate change is caused by human activities, not Earth’s natural warming and cooling cycle, said Gifford H. Miller, a geologist at the University of Colorado.

Cyclical warming is mostly related to the Earth’s irregular orbit around the sun,” he said. The Earth warms when it’s nearer the sun and cools when it’s farther away.

“For the past 10,000 years, we’ve been getting farther away,” he said. The exposure of such ancient moss suggests “the Arctic is now experiencing warmer summers than at any time since the end of the Ice Age.”


Carolina chickadees are trying to move north — like many other species dealing with climate change

Black-capped chickadees are commonly found in the Northeastern United States. Carolina chickadees make their home in the Southeast. Between them is a narrow zone in which both breeds reproduce in the spring. As winter temperatures have risen over the past decade, the birds’ social scene has moved steadily northward. Today, it is about seven miles farther north than it was in 2004.

The reason? Carolina chickadees are trying to move north — like many other species dealing with climate change — and are running into the black-caps.

“As they start interacting with the black-caps, they try to hybridize,” said Robert L. Curry, a biologist at Villanova University who has studied the birds. But a high percentage of the hybrid eggs don’t hatch, he has found, and hybrid chickadees are probably less fertile.

This is unfortunate for both species in the short term, but it would be even worse for two species not accustomed to mixing.

“This is a model for what could happen if you had an introduced species moving into a new area because of climate change, then come in contact with a species it’s never met before,” Dr. Curry said.


Wild flowers can’t focus

DOUGLAS QUENQUA

To everything there is a season — including, of course, the flowering of plants. But a warming climate is changing the timing in complicated ways. Scientists reviewed 39 years of records of flowering plants in the Rocky Mountains in Colorado, a period in which each decade saw a 0.72 degree Fahrenheit increase in average summer temperatures and a 3.5-day earlier spring snow melt.

In a study, published in Proceedings of the National Academy of Sciences last spring, found considerable variation in the changes in flowering, and a much larger number of species affected than previously believed.

Some form of flowering change occurred in 41 of 60 species examined. On average, first flowering advanced by 3.3 days per decade, peak flowering by 2.5 days, and final flowering by 1.5 days.

“The changes in the flower community are potentially reshuffling what’s available for the pollinators,” said a co-author, Amy M. Iler, a postdoctoral researcher at the Rocky Mountain Biological Laboratory. “We don’t know what all the consequences will be. It’s likely it will be good for some and bad for others.”


Fruit Flies Panic Because They Cannot Reproduce Efficiently

As temperatures rise, insect populations may relocate around the globe in search of more hospitable environments. But it is the extreme highs, not just the average rise in temperatures, that may determine where they end up.t-weight:400;”>Scientists studied 10 different fruit fly species in Australia (both temperate and tropical), noting the temperature ranges each preferred for mating and everyday life, and their thresholds for extreme hot and cold.

All the species lived in environments where temperatures were sometimes less than optimal, the researchers found, but none chose places that forced them to endure extreme heat.

“Many species might undergo seasons where conditions are not optimal for growth and reproduction,” said Johannes Overgaard, a biologist at Aarhus University in Denmark and an author of the study. “They just survive the season. But what they can’t survive is temperatures beyond their threshold.”

This is bad news for the insects in Australia, who might find themselves with fewer habitable lands as extreme conditions dominate the continent. Whether this will also hold true for other continents is not yet known, Dr. Overgaard said.


Rock Snot invasion

An unsightly algae known as “rock snot” has been surfacing in lake waters in Eastern Canada. “It looks like torn-up toilet paper that is attached to rocks,” said John Smol, a biologist at Queen’s University in Ontario who is studying the algae’s growth. “It’s an aesthetic issue, and as it decomposes it becomes a smell issue.”

Rock snot, or didymo, was thought of as an invasive species introduced by humans. But an analysis of fossilized algae in the lakes indicates that it is native.

The algae was present in one lake in Quebec since at least 1970, 36 years before it was first noticed, Dr. Smol’s team found.

Didymo tends to grow in flowing waters. Warmer winters may be producing less ice and snow that disrupt the flow.

Over time, the rock snot will become much more than an eyesore, Dr. Smol said. It will displace other organisms and destroy fish habitats.


A new type of Reef that thrives in high acidity

SINDYA N. BHANOO

Ocean acidification endangers coral in every ocean. But researchers have recently discovered unusual reefs in Palau that are thriving in increasingly acidicified waters. Ocean acidification occurs when carbon emitted by human activities mixeswith ocean waters. This decreases carbonate ions in the water, which coral andother organisms need to form their protective shells.

Yet in 2012, researchers working in the waters off Palau identified coralreefs that were both extremely acidified and very healthy. What’s differentabout these reefs, said Kathryn Shamberger, anoceanographer at Texas A&M University, is that the waters became acidified through natural means.

“The growth of the reef itself and the breathing of the organisms on the reef,” not man-made emissions, added carbon to the water, she said.


Shell Fish who can’t build shells

Increasing ocean acidity makes it difficult for marine species to build their shells and, by softening calcium carbonate, makes shells weaker. That’s bad news not only for clams, oysters and scallops, but for tens of thousands of lesser known species — echinoderms like star fish and sea urchins, colonies of tiny invertebrates, reef corals and many others.The Biological Bulletin devoted an issueto research on ocean calcification with papers and reviews on a large variety of organisms.

SINDYA N. BHANOO

Ocean acidification endangers coral in every ocean. But researchers have recently discovered unusual reefs in Palau that are thriving in increasingly acidicified waters. Ocean acidification occurs when carbon emitted by human activities mixeswith ocean waters. This decreases carbonate ions in the water, which coral andother organisms need to form their protective shells.

Yet in 2012, researchers working in the waters off Palau identified coralreefs that were both extremely acidified and very healthy. What’s differentabout these reefs, said Kathryn Shamberger, anoceanographer at Texas A&M University, is that the waters became acidified through natural means.

“The growth of the reef itself and the breathing of the organisms on the reef,” not man-made emissions, added carbon to the water, she said.


Shell Fish who can’t build shells

Increasing ocean acidity makes it difficult for marine species to build their shells and, by softening calcium carbonate, makes shells weaker. That’s bad news not only for clams, oysters and scallops, but for tens of thousands of lesser known species — echinoderms like star fish and sea urchins, colonies of tiny invertebrates, reef corals and many others.The Biological Bulletin devoted an issueto research on ocean calcification with papers and reviews on a large variety of organisms.

“Climate change and ocean acidification are going to manifest themselves in the ways species interact — eating each other, facilitating each other’s growth,” said an editor of the issue, Gretchen Hofmann, a marine biologist at the University of California, Santa Barbara.

And yet, she added, there is some hope. “In coastal areas there are plants that actually change the pH of the water — in a good way. Eel grass and surf grasses can provide refuge from future acidification.”

Where Do Acids in the Body Come From?

Excess Acidity Is Bad for Health and Contributes to All Diseases

I offer the above question as a “learning teaser.” The second of the above two statements is universally recognized and emphasized. As for the first statement, with rare exceptions, only diet is considered as source of acidity in the body. This is the most glaring example of neglect of real issues in my profession. Diet seldom, if ever, accounts for the main burden of total body acidity in chronic disease and unwellness.

In chronic illness,the main sources of excess acidity are:

  1. Molecular Biology of oxygen
  2. Gut ecology and gut microbiome
  3. Insulin homeostasis

The diet as the source of total body acidity is seldom a significant issue. Here is one of myriad ironies of the subject of acid-alkaline balance: People, even most doctors, attribute stomach conditions, such as gastritis, GERD, stomach and duodenal ulcers, esophagitis, and hiatal hernia, and heartburn (in common vernacular) as caused by acidity. The acidity in all of the above comes from disruptions of:

  1. Molecular Biology of oxygen
  2. Gut ecology and gut microbiome
  3. Insulin homeostasis

I recognize the above as medical ironies. Next I recognize the medical tragedies: The prevailing medical teaching and standards completely neglect the above three matters. The patients pay a large price, often for years, because of the ignorance and neglect of medical practitioners.

 


 

Three primary mechanisms of cellular injury in all chronic diseases in Ali GIO Model of Health and Healing are:

  1. Disrupted Molecular Biology of Oxygen,

  2. Disrupted Oxygen signaling, and

  3. Disrupted Insulin Homeostasis and Insulin Signaling


 

ALI GIO HEALING – STRONG SCIENTIFIC CLAIMS REQUIRE STRONG SCIENTIFIC EVIDENCE

The central claim of Ali GIO model is that the three primary mechanisms of cellular injury in all chronic diseases are:

  1. Disrupted Molecular Biology of Oxygen and perverted oxygen signaling

  2. Disrupted Insulin Homeostasis and perverted insulin signaling (insulin toxicity)

  3. Disrupted gut ecology and perverted degrees of gut fermentation (increased gut burden of gut flora.


 

The above three are serious scientific claims that I make in Ali GIO Healing and form the basis of my next three scientific claims:

  1. No treatment plans for healing any chronic disease can be considered complete unless all disrupted oxygen signaling is restored to normal oxygen signaling.

  2. No treatment plans for healing any chronic disease can be considered complete unless all disrupted insulin signaling is restored to normal oxygen signaling.

  3. No treatment plans for healing any chronic disease can be considered complete unless all disrupted patterns of gut ecology and gut microbiome are restored to normal gut ecology and gut microbiome.

ALI GIO HEALTH & HEALING COURSE

VIDEO LIBRARY

The left side panel of all parts of ALI GIO Health and Healing presents in alphabetical order a large library of the author’s videos on diverse subjects covered in the series of articles. Please select a video of your interest and click on the video listing to learn from the video. Links below take you to a series of videos on the subjects of gut ecology and microbiome and insulin toxicity.

Readers will find this series very informative and useful.

 

Bowel Health Series

  1.  Bowel-Colon Health videos part 1

https://majidalimd.me/bowel-videos/

2.  Bowel-Colon Health videos part 2

https://majidalimd.me/bowel-videos-part-2/

Insulin Toxicity Video Series

https://majidalimd.me/insulin-toxicity-and-dysfunction-videos-part-1/


ALI GIO Library of Published Articles and Book

  1. Ali M. Oxidative regression to primordial cellular ecology. J Integrative Medicine 1998; 2:4-55.
  2. Ali M. Bradford R, Ali O, et al. Immunostaining of Candida organisms in peripheral blood blood smears (abstract) 1995, American Academy of Otolaryngic Allergy, Spring Meeting, Palm Desert, CA.
  3. Ali M. Ramanarayanan MP:  A computerized micro-ELISA assay for allergen-specific IgE antibodies.  Am J Clin Pathol, 81:591-601, 1984.
  4. Ali M. Ramanarayanan MP, Nalebuff DJ, Fadal RG, Willoughby JW:  Serum concentrations of allergen-specific IgG antibodies in inhalant allergy: effect of specific immunotherapy.  Am J Clin Pathol, 80:290-299, 1983.
  5. Ali M. Respiratory-to-fermentative shift in immune-inflammatory disorders with energy deficit. Townsend Letter. 2004; 253;64-65.
  6. Ali M. Krebs cycle impairment – molecular biology of  oxygen. communication. Communication E-published. Nature. 2014;515:431.
  7. Ali M. Ali O. Oxidative coagulopathy in fibromyalgia and chronic fatigue syndrome. Am J Clin Pathol  1999; 112:566-7.Ali M. Krebs cycle impairment – molecular biology of  oxygen. communication. Communication E-published. Nature. 2014;515:431.
  8. Ali M. Oxygen and Aging. . (2nd ed.) New York, Canary 21 Press. 2004.
  9. Ali M. Spontaneity of oxidation in Nature and Aging. Monograph). Teaneck, New Jersey, U.S.A. 1983.
  10. Ali M. Hurt human habitat and energy deficit—Healing Through Restoration of Krebs cycle chemistry. Townsend Letter-The examiner of Alternative Medicine. 2006; 279:112-115.
  11. Ali M. The Principles and Practice of Integrative Medicine Volume  III: Pathobiology by Micro-Ecologic Cellular and Macro-Ecologic Tissue-Organ Systems 1999.  New York. Canary 21 Press. 1999.
  12. Ali M. The Principles and Practice of Integrative Medicine Volume  IV: Pathobiology by Micro-Ecologic Cellular and Macro-Ecologic Tissue-Organ Systems 1999.  New York. Canary 21 Press. 1999.
  13. Ali M. Altered States of Bowel Ecology. Monograph. Teaneck, New Jersey.1980.
  14. Ali M. The dysox model of aging.  Townsend Letter for Doctors. and Patients.2005;269:130-134.
  15. Ali M. The dysox model of aging.  Townsend Letter for Doctors and Patients.2005;269:130-134.
  16. Ali M. Obesity is cellular oxygen defunction state. Aging Healthfully Healthfully.  2004;5:2-19.
  17. Ali M. The oxidative-dysoxygenative perspective of allergic disorders. J Integrative Med. 2000; 4:1-17.
  18. Ali M. Beyond insulin resistance and syndrome X: The oxidative-dysoxygenative insulin dysfunction (ODID) model. J Capital University of Integrative Medicine. 2001;1:101-141.
  19. Ali M. The Dysox Model of Diabetes and De-Diabetization Potential. Townsend Letter-The examiner of Alternative Medicine. 2007; 286:137-145.
  20. Ali M. Oxygen, Insulin Toxicity, Inflammation, and  the Clinical Benefits of Chelation. Part I. Townsend Letter-The examiner of Alternative Medicine. 2009;315:105-109. Ali M. Insulin Reduction and EDTA Chelation: Two Potent and Complementary Approaches For Preventing and Reversing Coronary Disease. Oxygen, Insulin Toxicity, Inflammation, and the  Clinical Benefits of Chelation – Part II. Townsend Letter-The examiner of Alternative Medicine. 2010;323:74-79. June 2010.
  21. Ali M. Epidemic of Dysoxygenosis and the Metabolic Syndrome. In: The Principles and Practice of Integrative Medicine. Volume 5. Pp 246-256. Canary 21 Press. New York. 2005.
  22. Ali M. Insulin: the life span hormone for healthful aging. Townsend Letter. The Examiner of Alternative Medicine . 2018;416:54-59.
  23. Ali M. Death begins in the colon. Ali Academy.  http://aliacademy.org/problems_of_the_gut.htm
  24. Ali M. As roots are to roses, so the bowel to the brain. Diseaases begin in the fermenting colon.  Ali Academy.http://aliacademy.org/problems_of_the_gut.htm
  25. ‘Ali M. Ali M. Seed, Feed, and Weed Plan for Restoring Gut Flora   and Metabolic Health. Communication e-published in Nature. 2016;535:56-64.
  26. Chouchani ET, Pell VR, Guade E, et al. Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS. Nature.2014;515:431-435.
  27. Ali M. The agony and death of a cell. In: Syllabus of the Instruction Course of the American Academy of Environmental Medicine. Denver, Colorado, 1985.
  28. Ali M.The Principles and Practice of Integrative Medicine Volume X: Darwin, Oxygen Homeostasis, and  Oxystatic Therapies.  3 rd. Edi. (2009) New York. Institute of Integrative Medicine Press.
  29. Ali M. The Principles and Practice of Integrative Medicine Volume  XI: Darwin, Dysox, and Disease. 2000. 3rd. Edi. 2008. New York.  (2009) Institute of Integrative Medicine Press.
  30. Ali M. The Principles and Practice of Integrative Medicine Volume  XII: Darwin, Dysox, and Integrative Protocols. New York (2009). Institute of Integrative Medicine Press.
  31. Ali M. Petrochemical Illness and philic-phobic dysequilibrium in the Gulf of Mexico. Townsend Letter-The examiner of Alternative Medicine. 2010;76:79. November 2010.
  32. Ali M. Oxyphilic Nature Ali M. Fungal Infections and Oxyphile-Oxyphobic Conflicts. Communication e-published Nature. 2017.
  33. Ali M. The dysox model of renal insufficieny and improved renal function with oxystatic therapies. Townsend Letter for Doctors and Patients.2005;267:101-108.
  34. Ali M. Fischer S, Juco J, et al. The dysox model of coronay artery disease. Townsend Letter for Doctors and Patients. 2006;270/71:110-112.
  35. Ali M. The ADHD-Autism-Oxygen Connection: The Larger-Head-Smaller-Brain Scenario. Townsend Letter-The examiner of Alternative Medicine. April, 2008. http://www.townsendletter.com.
  36. Ali M. Oxygen model of Alzheimer’s disease. In . Oxygen and Aging. (2nd ed.) New York, Canary 21 Press. 2004.
  37. Ali M. Darwin Moms, Nursing Milk, and Antibiotic Resistance. In: Nature; 2016. 533:212.
  38. Progenitor stem cell progression model of autism (PSP Autism model). spectrum WordPress. 2016.
  39. Ali M.  Osteocrin, making connections, and autism. Communication e-published. Nature. 2016;539:242.
  40. Ali M. Chronic disease is evolution in reverse. Communication e-published. Nature. 2016. 530:268.
  41. Ali M. The Principles and Practice of Integrative Medicine Volume I: Nature’s Preoccupation With Complementarity and Contrariety. New York. Canary 21 Press. 1998. 2nd edition  2005.
  42. Ali M. The Principles and Practice of Integrative Medicine Volume  II: The History and Philosphy of Integrative Medicine. 2001.  New York. Canary 21 Press. 1998. 2nd edition  2006.
  1. Ali M. The Principles and Practice of Integrative Medicine Volume  III: Pathobiology by Micro-Ecologic Cellular and Macro-Ecologic Tissue-Organ Systems 1999.  New York. Canary 21 Press. 1999
  2. Ali M. The Principles and Practice of Integrative Medicine Volume  IV: Integrative Immunology and Allergy: The Oxidative-Dysoxygenative Perspective.  New York. Canary 21 Press. 1998. 2nd edition  2005.
  3. Ali M. The Principles and Practice of Integrative Medicine Volume  V: Integrative Nutritional Medicine: Nutrition Seen Through the Prism of Oxygen Homeostasis.  New York. Canary 21 Press. 1999. 2nd edition  2005.
  4. Ali M. Oxidative cell membrane disorder – Leaky Cell Membrane Disorder (monograph). Teaneck, NJ, 1987.
  5. Ali M. Darwin, oxidosis, dysoxygenosis, and integration. J Integrative Medicine 1999;3:11-16.
  6. Ali M:  Molecular basis for the use of antioxidants to regulate plasma membrane dynamics in allergic disorders.  abstracts, Annual Meeting of the American Academy of Otolaryngic Allergy, page 30, 199l.
  7. Ali M: Darwin, fatigue, and fibromyalgia.J Integrative Medicine 1999;3:5-10.
  8. Xu G, Buyun, SL, Buyun. Corrected prevalence of autism spectrum disorder among US children and adolescent. JAMA. 2018;319:505.
  9. He L, Si G, Huang J, et al. Mechanical regulation of stem-cell differentiation by the stretch-activated Piezo channel. Nature. 2018;555:103-106.
  10. intestinal stem cell xxxxxxxxx xxxxxx / https://alihealing.org/2018/10/02/what-might-autism-teach-about-alzheimers-disease-ad-what-might-ad-teach-about-autism/

 

  1. Noctor SC, Martinez-Cerdeno V, Kriegstein AR. Contribution of intermediate progenitor cells to cortical histogenesis. Arch. Neurol. 2007;64:639-642.
  2. Seaberg RM, Van Der Kooy D. Stem and Progenitor cells. The premature desertion of rigorous definitions. Trends in Neurosciences. 2003;26:125-131.

56.Toriumi K, Mouri A, Narusawa S, et al. Prenatal NMDA Receptor Antagonism Impaired Proliferation of Neuronal Progenitor, Leading to Fewer Glutamatergic Neurons in the Prefrontal Cortex. Neuropsychopharmacology.2012;37: 1387-139./

  1. 57. Krakowiak P, Walker CK, Bremer AA, et al. Maternal metabolic conditions and risk for autism and other neurodevelopmental disorders. Pediatrics2012; 129(5): e1121–e1128.
  2. Bilbo SD, Tsang V. Enduring consequences of maternal obesity for brain inflammation and behavior of offspring. FASEB J 2010; 24(6): 2104–2115.
  3. Catalano P. The impact of gestational diabetes and maternal obesity on the mother and her offspring. J Dev Orig Health Dis2010; 1(04): 208–215.
  4. Van Lieshout R, Taylor V, Boyle M. Pre‐pregnancy and pregnancy obesity and neurodevelopmental outcomes in offspring: a systematic review. Obes Rev 2011; 12(5): e548–e559.
  5. Kosidou K,Kalman C, Gardner RM. Maternal Polycystic ovary syndrome and the risk of Autism spectrum disorders in the offspring. A population-based nationwide study in Sweden. Molecular Psychiatry.21:1441-144b.
  6. Cheslack-Postava K, Fallin MD. Beta adrenergic receptor gene variant and risk of autism in the AGRE cohort. Molecular Psychiatry. 2007;12:283-81.
  7. 63. Yazawa M, et al.Using induced pluripotent stem cells to investigate cardiac phenotypes in Timothy syndrome. Nature 471, 230–234 (2011).
  8. Sugimura R, Jha DK, Han Areum et al.  Haematopoietic stem and progenitor cells from human pluripotent stem cells. Nature. 2017;545;432-38.
  9. Lancaster, M. A. et al.Cerebral organoids model human brain development and microcephaly. Nature.2013;501, 373–379.
  10. Ataman B, Boulting G, Harmin DA, et al. Evolution of Osteocrin as an activity-regulated factor in the primate brain. Nature.  2016; 539: 242-247.
  11. Canter RG,Penney J, Tsai L-H. The road to restoring neural circuits for the treatment of Alzheimer’s disease. Nature. 2016;539:187-196.
  12. Mardinly, A. R. et al.Sensory experience regulates cortical inhibition by inducing IGF1 in VIP neurons. Nature. 2016;531, 371–375.
  13. Quadrato G, Nguyen T, Macosko EZ, et al. Cell diversity and network dynamics in photosensitive human brain organoids. Nature. 2017;545:48-53.
  14. Birey F. Anderson J, Makinson CD. Et al.Assembly of functionally integrated human forebrain spheroids. Nature.2017;545:54-59.

71.Sweatt D, Layered-up regulation in the development developing brain.  Communication e-published.Nature. 2017;551:448-9.

  1. Kapp FG, Perlin JR, Hagedorn EJ, et al. Protection from UV light is an evolutionarily conserved feature of the haematopoietic niche. Nature. 2018;558:445-448.
  2. Lin G, Xu N, Xi, Paracrine signaling controls self-renewal of Drosophila intestinal stem cells.. Nature.2008;455.
  3. Ali M. Epigenetic and Clinical Implications of Neural Spheroids and Organoids In Treating Autism.. Communication. E-Published. Nature. 2017;545:54-59.
  4. Ali M Unpacking Brain Organoids and Spheroids for Delineating the Impeded Progenitor Cell Progression Model of the Autism Spectrum Disorder. Communicaion. E-Published. Nature. 2017; 545:48-53.
  5. Ali M.  Genetics and Comorbidities of the Autism Spectrum disorder. Communicaion. E-PublishedNature. 2017;543:507-512.
  6. Ali M.  Integration of Empirical Measures for Brain  Rejuvenation. Communicaion. E-Published.Nature. 2016;539:187.
  7. Ali M. Osteocrin, Making Connections, and Autism.  Communication e-published. Nature. 2016;539:242.
  8. Ali M.  Genetics and Comorbidities of the Autism Spectrum

disorder. Communicaion. E-Published Nature. 2017;543:507-512.

  1. Ali M.  Integration of Empirical Measures for Brain  Rejuvenation. Communication e-published. Nature. 2016;539:187.
  2. Ali M.Tribal Brain Model and Autism. Communication e-published. Nature. 2018;554:364-367.
  3. Ali M. Enteroendocrine Stem cells and Two Dimensions Enteroendocrine Dimension of the Autism Spectrum Disorder. Nature. 2018;555:103-106.
  4. Ali M. Epigenetic and Clinical Implications of Neural Spheroids and Organoids for integrative Management of Autism. Communication e-published in Nature. Nature.2017;545:54-59.
  5. Ali M. Hypothalamic Influences on Metabolism, Feeding, and Insulin. Nature. 2016;531:647-650.
  6. Ali  M. Irritable Bowel Syndrome and Seed,Feed, and Weed Approach. Communication e-published. Nature.  2016;533.
  7. Ali M. Darwin Moms , Nursing Milk, and Antibiotic Resistance. Communication e-published. Nature.2017;533:212.
  8. Ali M.Maternal Gut Microbes, Immune Connections, and Autism Circuits. Comment. E-Published. Nature. 2018; 549:528–532 & . 482-487.
  9. Ali M. Women-Like Men and Men-Like Women . Nature 533:206.
  10. Ali M.  Menstruating Mice and Human Reproductive Health. Nature. 2016.June.
  11. Ali M. 30. Workable Simplicity for Coping With Complexity of Diabetes. Nature.2016;534: 447
  12. Ali M. Reversing Diabetes – Acetate Mediates Metabolic   Syndrome. Nature.2016;534: 213.
  13. Ali M.
  14. Ali M. Recognized Empirtical Benefits of Methylation – What Next? Communication               e-published.Nature. 2016;369:373.
  15. Ali M. Darkfield Ali M. The Crab, Oxygen and Cancer. Volume I: The Dysox Model of Cancer.  2007.  New York, Canary 21 Press. Pages 114-116.
  1. Ali M. Cell Membrane Dysregulation. Syllabus. American Academy of Environmental Medicine. 1988. 13th Instructional Course. Cleveland, Ohio. pp 109-137.
  2. https://www.cdc.gov/mold/stachy.htm.
  3. Bitnun A, Nosal RM. Stachybotrys chartarum (atra)contamination of the indoor environment: Health implications. Paediatr Child Health. 1999;4:125-129.
  4. Shaw, W., et. al. Increased Urinary Excretion of Analogs of Krebs Cycle Metabolites and Arabinose in Two Brothers with Autistic Features. Clin Chem 41:1094-1104, 1995. 99. Shaw, W., et. al. Assessment of antifungal drug therapy in autism by measurement of suspected microbial metabolites in urine with GC/MS. Clinical Practice of Alternative Medicine: 15-26.
  5. https://alihealing.org/2018/10/02/what-might-autism-teach-about-alzheimers-disease-ad-what-might-ad-teach-about-autism/
  6. Ali M. Succinate Retention. In: Chouchani ET, Victoria R. Pell VR, Edoardo Gaude E, et. al. Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS. Nature. 2014;515:431–435.
  7. Ali M. Succinate Retention: The Core Krebs Dysfunction in Immune-Inflammatory Disorders. Townsend Letter. 2015;388:84-85.
  8. Ali M: Darwin, oxidosis, dysoxygenosis, and integration. J Integrative Medicine 1999;3:11-16.
  9. Climatic Chaos –  The primacy of oxygen issues over carbon issues. Part I. Townsend Letter-The examiner of Alternative Medicine. 2008;299:125-132.
  10. Ali M. The Principles and Practice of Integrative Medicine Volume X: Darwin, Oxygen Homeostasis, and  Oxystatic Therapies.  3 rd. Edi. (2009) New York. Institute of Integrative Medicine Press.
  11. Ali M. The Principles and Practice of Integrative Medicine Volume  XI: Darwin, Dysox, and Disease. 2000. 3rd. Edi. 2008. New York.  (2009) Institute of Integrative Medicine Press.
  12. Ali M. Epidemic of Dysoxygenosis and the Metabolic Syndrome. In: The Principles and Practice of Integrative Medicine. Volume 5. Pp 246-256. Canary 21 Press. New York. 2005.
  13. Ali M. Dysox Model of Diabetes and De-Diabetization Potential. Townsend Letter-The examiner of Alternative Medicine. 2007; 286:137-145.
  14. Ali Plan for Reversing Diabetes. New York, Canary 21 Press. Aging Healthfully Book 2011.
  15. Ali M. Oxygen, Insulin Toxicity, Inflammation, and  the Clinical Benefits of Chelation. Part I. Townsend Letter-The examiner of Alternative Medicine. 2009;315:105-109. October, 2009.
  16. Ali M. Fayemi AO, Ali O. Dasoju S, et al. Shifting Focus From Glycemic Status to Insulin Homeostasis. .  Townsend Letter-The Examiner of Alternative Medicine. 2017;402:91-96.
  17. Ali M. Insulin Reduction and EDTA Chelation: Two Potent and Complementary Approaches For Preventing and Reversing Coronary Disease. Oxygen, Insulin Toxicity, Inflammation, and the  Clinical Benefits of Chelation – Part II. Townsend Letter-The examiner of Alternative Medicine. 2010;323:74-79. June 2010.
  18. Ali M. Fayemi AO, Ali O. Dasoju S, et al. Shifting Focus From Glycemic Status to Insulin Homeostasis. .  Townsend Letter-The Examiner of Alternative Medicine. 2017;402:91-96.
  19. Ali M. Oxidative Cell Membrane Disorder – Leaky Cell Membrane Disorder (monograph). Teaneck, NJ, 1987.
  20. Kahn SE, 1, Hull RL, Utzschneider KM. Mechanisms linking obesity to insulin resistance and type 2 diabetes. Nature 2006;444, 840-846.
  21. Shoelson SE, Lee J, Goldfine AB. Inflammation and insulin resistance. J Clin Invest. 2006;116 :1793B1801.
  22. Shulman G. Ectopic Fat in Insulin Resistance, Dyslipidemia, and Cardiometabolic Disease. N Engl J Med. 2014; 371:1131‑
  23. International Diabetes Federation. Diabetes Atlas. 2016. Seventh edition. diabetesatlas.org.
  24. Sako, Y. & Grill, V. E. A 48-hour lipid infusion in the rat time-dependently inhibits glucose-induced insulin secretion and B cell oxidation through a process likely coupled to fatty acid oxidation. Endocrinology127, 1580–1589 (1990).|
  25. Kahn, S. E., Bergman, R. N., Schwartz, M. W., Taborsky, G. J. & Porte, D. Short-term hyperglycemia and hyperinsulinemia improve insulin action but do not alter glucose action in normal humans.  J. Physiol.1992;262: E518–E523.
  26. Ali M. Ali M. The Principles and Practice of Integrative Medicine Volume  III: Pathobiology by Micro-Ecologic Cellular and Macro-Ecologic Tissue-Organ Systems 1999.  New York. Canary 21 Press. 1999.
  27. Ali M, Braun EV, Fayemi AO:  Pathology.  7th ed.  New York, Med Exam, 1980,370 pp.
  28. Ali M. The dysox model of aging.  Townsend Letter for Doctors and Patients.2005;269:130-134.Aging
  29. Ali M, Ramanarayanan MP, Nalebuff DJ, Fadal RG, Willoughby JW:  Serum concentrations of allergen-specific IgG antibodies in inhalant allergy: effect of specific immunotherapy.  Am J Clin Pathol, 80:290-299, 1983.
  30. Ali M. Spontaneity of Oxidation in Nature and Aging,  (monograph). Teaneck, NJ, 1983.
  31. Ali M, Ramanarayanan MP:  A computerized micro-ELISA assay for allergen-specific IgE antibodies.  Am J Clin Pathol, 81:591-601, 1984.
  32. Ali M.   Hyperinsulinism Associated With Breast and Prostate Cancer. Townsend Letter-The Examiner of Alternative Medicine.2017.
  33. Ali M. Altered States of Biology Ecology. (monograph). Teaneck, NJ, 1980.
  34. Murphy KG, Bloom SR. Gut hormones and the regulation of energy bhomeostasis. 2006;444:854-859.
  35. Ali M. Dysox Model of Cancer Cancer
  36. Philosophy and Science of holism in healing. APPNA Journal. 2015.
  37. Ali M. Hyperinsulinism Associated With Breast and Prostate Cancer. Townsend Letter-The Examiner of Alternative Medicine.2017.
  38. Ali M. Molecular Basis of Autism and Dysuatonomia – The Impeded Progenitor Cell Progression (IPCP) model of ASD and Dysautonomia. Townsend Letterfor Doctors and Patients. 2017 (In press)
  39. Ali M. The Principles and Practice of Integrative Medicine Volume X: Darwin, Oxygen Homeostasis, and  Oxystatic Therapies.  3 rd. Edi. (2009) New York. Institute of Integrative Medicine Press.
  40. Ali M. The Principles and Practice of Integrative Medicine Volume  XI: Darwin, Dysox, and Disease. 2000. 3rd. Edi. 2008. New York.  (2009) Institute of Integrative Medicine Press.
  41. Ali M. The Principles and Practice of Integrative Medicine Volume  XII: Darwin, Dysox, and Integrative Protocols. New York (2009). Institute of Integrative Medicine Press.
  42. Insulin Laboratory Ranges. http://alidiabetes.org/2016/02/25/insulin-laboratory-ranges/
  43. Ali Importance of Subtyping Diabetes Type 2 Into Diabetes Type 2A and Diabetes Type 2B. Townsend Letter-The Examiner of Alternative Medicine. 2014; 369:56-58.
  44. Ali M. Dasoju S, Karim N, Amin J, Chaudary D. Study of Responses to Carbohydrates and Non-carbohydrate Challenges In Insulin-Based Care of Metabolic Disorders. Townsend Letter-The Examiner of Alternative Medicine. 2016; 391:48-51.
  45. Ali M. Integrative Cardiology and Chelation Therapies. The Principles and Practice of Integrative Medicine. Vol VI. . (2005) New York. Institute of Integrative Medicine Press.
  46. Fontoura P. Harnessing Emerging Science for Unmet Needs patient needs. In: Sponsor Feature. Nature. 2016;491, Preceding page S1.
  47. Wall DP, Esteban FJ, Deluca TF et al. Comparative analysis of neurological disorders focuses genome-wide search for autism genes. Genomics. 2008;93(2):120–9.

ALI GIO HEALTH & HEALING COURSE

VIDEO LIBRARY

The left side panel of all parts of ALI GIO Health and Healing presents in alphabetical order a large library of the author’s videos on diverse subjects covered in the series of articles. Please select a video of your interest and click on the video listing to learn from the video. Links below take you to a series of videos on the subjects of gut ecology and microbiome and insulin toxicity.

Readers will find this series very informative and useful.

 

Bowel Health Series

  1.  Bowel-Colon Health videos part 1

https://majidalimd.me/bowel-videos/

2.  Bowel-Colon Health videos part 2

https://majidalimd.me/bowel-videos-part-2/

Insulin Toxicity Video Series

https://majidalimd.me/insulin-toxicity-and-dysfunction-videos-part-1/

END END

 

Leave a Reply

Fill in your details below or click an icon to log in:

WordPress.com Logo

You are commenting using your WordPress.com account. Log Out /  Change )

Google photo

You are commenting using your Google account. Log Out /  Change )

Twitter picture

You are commenting using your Twitter account. Log Out /  Change )

Facebook photo

You are commenting using your Facebook account. Log Out /  Change )

Connecting to %s