Majid Ali, M.D.
By 2025, the cost of treating Alzheimer’s disease in the United States is estimated to reach $1 trillion. Where will the country get this much money? What kind of care will that money buy in 2030? No clear answers to these questions are forthcoming – unless you turn to spices.
Researchers at Vanderbilt University recently stated:
“One of the most promising new treatments for Alzheimer’s disease may already be in your kitchen. Curcumin, a natural product found in the spice turmeric, has been used by many Asian cultures for centuries, and a new study indicates a close chemical analog of curcumin has properties that may make it useful as a treatment for the brain disease.
“Curcumin has demonstrated ability to enter the brain, bind and destroy the beta-amyloid plaques present in Alzheimer’s with reduced toxicity,” said Wellington Pham, Ph.D., assistant professor of Radiology and Radiological Sciences and Biomedical Engineering at Vanderbilt and senior author of the study, published recently in the Journal of Alzheimer’s Disease.
Accumulation and aggregation of protein fragments, known as beta-amyloid, drives the irreversible loss of neurons in Alzheimer’s disease. The rest of the article discusses the use of curcumin to get drugs for Alzheimer’s across the brain-blood barrier.
Below are suggestions that we have been publishing for years – including a spicy breakfast and spice enriched meals.
What can individuals do to reduce the chance of developing Alzheimer’s disease?
- At the fundamental level, Alzheimer’s disease is caused by oxygen deficit in the brain. So the way to prevent the disease is to “think oxygen” and use the brain to keep its cells breathing their oxygen.
- At the next level, anything that causes inflammation anywhere in the body increases the risk of Alzheimer’s disease. So the way to prevent the disease is to “think inflammation” and take steps to prevent inflammation in the body.
- At another level, nutrient deficits reduce oxygen in the brain and increase inflammation in the body and so increase the risk of Alzheimer’s disease. So the way to prevent the disease is to “think nutrients” and provide the body with the needed nutrients.
- At another level, chronic stress robs the brain of oxygen, increases acidity and free radical activity in it, and increases the risk of Alzheimer’s disease. So the way to prevent the disease is to “think personal spiritual realism” and reduce exposure to chemicals.
- At another level, physical inactivity slows blood circulation in the body, including in the brain. So the way to prevent the disease is to “think fitness” and take long walks.
- At another level, all synthetic chemicals cause inflammation in the body and so increase the risk of Alzheimer’s disease. So the way to prevent the disease is to “think chemicals” and reduce exposure to chemicals.
- At another level, toxic metals (mercury, lead, aluminum, and others) directly injure the energy systems in the brain cells. So the way to prevent the disease is to “think toxic metals” and remove them with chelation.
- At another level, spices reduce inflammation in the body and so decrease the risk of Alzheimer’s disease. So the way to prevent the disease is to “think spices.” Dr. Ali’s Spicy breakfasts.
- And read and write, sing and dance, be compassionate to others – and to yourself.
Alzheimer’s disease (AD) is a condition of brain cell loss that results in shrinking of the brain. It robs the person of his personality, memory, judgment, dignity, and identity. AD leaves its victims helpless , leaves them and hapless, unable to care for themselves or anyone else. Men and women with AD lose their spouses, and their spouses lose them. Alzheimer’s disease usually takes lives of individuals affected by it at a slower rate than most cancers that return after surgery. So, it is the most heart-wrenching problem faced by the family members who care for the individual.
What causes Alzheimer’s disease?
Is it bad diet? Yes, there is considerable evidence to support that view. Could it be deficiencies of essential nutrients? Yes, there is evidence for that as well. Could it be tobacco smoking? Yes, that too. How about lead and mercury toxicity? Certainly. Do many environmental pollutants injure brain cells and destroy them? Undoubtedly.
Do hypertension, obesity, and diabetes increase the risk and prevalence of Alzheimer’s disease?
There is no doubt of that.
Do chronic inflammatory and immune disorders increase the prevalence of the disease?
Does emotional stress at home lead to memory loss? And work-related stress?
And the quality of drinking water? Air quality? And how might habitual mental laziness affect the memory function? There is, of course, evidence for that?
Are strokes associated with a higher risk of Alzheimer’s disease? How often does chemotherapy diminish memory status? Do coronary bypass operations increase the risk of memory loss? And autoimmune disorders? And inflammatory disorders? The answer to all these questions is clearly yes.
Here is the crucial question: Can you think of some basic molecular-energetic defect that links the risk and prevalence of Alzheimer’s disease with all of the above facts? Yes. It is dysoxygenosis—dysox for short, dysfunctional oxygen metabolism for general readership.
We need oxygen!
Toxicities of foods, environment, and thoughts (disappointments, chronic anger, and mental health issues) impede or block the energetic, detergent, and developmental functions of oxygen. Dysfunctional oxygen metabolism (dysox) so produced causes injury and loss of brain cells. This is the beginning of inflammation, deposition of amyloid proteins, and plaques formation that are the hallmarks of Alzheimer’s disease (AD). This is the scientific basis of my Oxygen Model of Alzheimer’s disease.
Medical models are proposed to reduce complexities in biology to workable simplicities and to predict clinical characteristics of diseases.
One of the crucial predictions of the Oxygen Model of Alzheimer’s disease was validated in April 2011 with the publications of two large studies in the Journal Nature Genetics. An analysis of genes of more than 50,000 people in the United States and Europe doubled the number of genes—from five to ten— that increase the risk of Alzheimer’s disease. All ten are involved with inflammation and cholesterol metabolism. Cholesterol, of course is an antioxidant and, like other antioxidants in physiologic ranges in human biology, serve as anti-inflammatory roles and favorably influence molecular traffic across cell membranes.
Later on February 1, 2012, The New York Times published an article entitled “Path Is Found for the Spread of Alzheimer’s.” The writer, Mr. Gina Kolata, reported that “Alzheimer’s disease seems to spread like an infection from brain cell to brain cell, two new studies in mice have found. But instead of viruses or bacteria, what is being spread is a distorted protein known as tau.” Based on that, Mr. Kolata made a remarkable claim of a breakthrough.
An Antibody Cure?
Consider his wild enthusiasm in the following sentence: “The new studies provide an answer. And they indicate it may be possible to bring Alzheimer’s disease to an abrupt halt early on by preventing cell-to-cell transmission, perhaps with an antibody that blocks tau.” It is safe to predict that an antibody drug, when developed on this promise, will at best offer minimal and temporary benefits. My reason is that the tau protein abnormalities develop as a consequence of toxicities of foods, environment, and thoughts. No antibody drug can address these problems.
Cellular and Molecular Environments Do Not Matter
Gina Kolata ends his article with the following astonishing sentence: “It isn’t a bad neighborhood. It is contagion from one neuron to another.” Translation: toxicities of nutrition, environment, and thoughts do not matter in the cause of Alzheimer’s disease.
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Memory Loss – The Problem and Solution Seminar by Professor Majid Ali ONLY 9.95
In this 35-minute video seminar, Professor Majid Ali, M.D. describes how memory difficulties and loss begins and progresses. He explains the common error of ignoring brain nutrients that are essential for memory functions and toxicities of food, environment, and stress that interfere with neuronal functions. Notable among memory nutrients are vitamins B12 and other B-complex members, glutathione, taurine, and minerals (especially magnesium, potassium, and calcium). Notable among the toxicities are toxins produced by fermentation in the gut (colon, small intestine, stomach, esophagus, and the oral cavity. He then offers his guidelines to address these issues.