Dr. Ali’s Course on Allergy


Dr. Ali’s Course on Allergy

During the early 1980s, many clinical observations led me to investigate the role of altered states of the bowel ecosystem on the clinical manifestations of allergy and hypersensitivity reactions.

  • For example, a marked improvement in symptoms of atopic dermatitis was observed in many patients with empirical therapies that putatively “restored the bowel health.”
  • Relief of constipation was associated with relief of sinusitis headache in others.
  • Symptoms of allergic rhinitis often subsided with antifungal therapies.

Such observations led me to introduce the concept of altered states of bowel ecology as the basis for heightened hypersensitivity states.

Understanding allergy and the environment

Notwithstanding the great advances in both classical allergy and clinical ecology, certain fundamental aspects of clinical hypersensitivity reactions remained unexplained.

  • For instance, eczema lesions flare more in some weeks than in others in the same person.
  • Lifestyle stressors exaggerate bronchospasm more on some days than on others in the same asthma sufferer.
  • Symptoms of Crohn’s colitis and ulcerative colitis remit and relapse for no apparent reason in most instances.
  • Food sensitivity reactions vary over a broad range in the same individual.
  • Allergic rhinitis becomes more intense on some days when pollen count is low and abates on days when pollen counts are high.
  • The phenomenon of “spreading sensitivity reactions” (long vigorously denied by IgE researchers) is increasingly recognized in chronic fatigue syndrome, fibromyalgia, and multiple chemical sensitivity syndrome.

Adjunctive Nutrient, Herbal, and Oxygenative Therapies

The clinical outcome in allergy treatment is markedly enhanced if issues of lifestyle, stress and the ecologic integrity of the bowel, blood, and liver ecosystems are addressed. This becomes a critical issue when allergy symptomatology is a major part of diverse chronic immune disorders such as fibromyalgia, Crohn’s colitis, multiple sclerosis, and multiple chemical sensitivity syndrome.

My colleagues and I prescribe nutrient, herbal, and oxygenative protocols for restoring the various ecosystems of the body for individual patients in light of the total clinical context, rather than blinded and controlled trials of individual therapeutic agents. The latter approach, we have pointed out earlier, negates the very essence of the integrative approach to clinical problems.

Table 1. General Guidelines for Nutrient and Redox-Restorative Supplements for Atopic Patients With and Without Indolent Immune Disorders

Atopic Individuals Without Chronic Indolent Immune Dysfunction

Atopic Individuals With Chronic Indolent

Immune Dysfunction

Vitamins

Vitamins:

C, 1,000 to 2,000 mg;

E, 200 to 400 IU;

A, 5,000 to 7,500 IU;

D, 100 to 250 IU;

B-complex, 25-50 mg;

B12, 1,000 mcg weekly for four weeks.

Vitamins:

C, 3,000 to 5,000 mg;

E, 400 to 800 IU;

A, 10,000 to 15,000 IU;

D, 100 to 250 IU;

B-complex, 30-50 mg;

B12, 1,000 to 5,000 mcg weekly for four to six weeks .

Minerals

Magnesium, 1,000 to 1,500 mg;

Calcium, 750 to 1,000 mg;

Potassium, 200 to 400 mg;

Chromium, 100-300 mcg;

Selenium, 100-300 mcg;

Molybdenum, 100-300 mcg.

Magnesium, 1,500 to 2,500 mg;

Calcium, 1,000 to 1,500 mg;

Potassium, 400 to 600 mg;

Chromium, 400-600 mcg;

Selenium, 400-600 mcg;

Molybdenum, 400-600 mcg.

Redox-Restorative Substances

Glutathione, 200-300 mg;

N-acetylcysteine, 200-300 mg; Methylsulfonylmethane, 200 to 500 mg; Lipoic acid, 100 to 200 mg; Taurine, 500 to 1,000 mg; Coenzyme Q10, 30 to 50 mg;

Pycnogenol, 50 to 100 mg.

Glutathione, 600-800 mg;

N-acetylcysteine, 600-800 mg; Methylsulfonylmethane, 1,000 to 1,500 mg; Lipoic acid, 300 to 500 mg; Taurine, 1,500 to 2,000 mg; Coenzyme Q10, 100 to 150 mg;

Pycnogenol, 100 to 150 mg.

Table 2. Composition of Adjunctive Nutrient and Herbal Protocols for the Bowel

BOWEL ECOLOGY PROTOCOL #1

One billion spores of Lactobacillus acidophilus, Lactobacillus bulgaricus, Bifidobacterium in a base of complex vegetable fiber, magnesium sulfate, vitamin B complex, l-histidine, l-arginine, pantethine, aloe vera.

BOWEL ECOLOGY PROTOCOL #2

Alfalfa, 500 mg; pau d’arco,100 mg; allium, 100 mg; licorice root extract, 200 mg.

BOWEL ECOLOGY PROTOCOL #3

Calcium caprylate, 50 mg; magnesium caprylate,50 mg; grapefruit seed extract, 25 mg; aloe vera, 1 mg; spirulina, 10 mg.

BOWEL ECOLOGY PROTOCOL #4

Grapefruit seed extract, 50 mg; allium, 50 mg; pau d’arco, 500 mg.

BOWEL ECOLOGY PROTOCOL #5

Par-quing, 150 mg; pau d’arco 150 mg; beet root fiber, 200 mg; guar gum, 100 mg.

BOWEL ECOLOGY PROTOCOL #6

Echinacea, 200 mg; goldenseal root, 150 mg; astragalus root, 150 mg; burdock root 150 mg.

Table 3. Composition of Intramuscular Protocol A

Nutrient

Concentration

Volume

Magnesium sulfate.

500 mg/ml

1.5 ml

Calcium glycerrhate/lactate

10 mg/ml

1.5 ml

Vitamin B12

10,000 mcg/ml

0.5 ml

Vit.B complex

*

1 ml

Pantothenic acid

250 mg/ml

0.5 ml

Pyridoxine

100 mg/ml

0.5 ml

Zinc

5 mg/ml

0/6 ml

Molybdenum

25 mcg/ml

0.5 ml

Selenium

40 mcg/ml

0.4 ml

Multivitamin

*

0.5 ml

* Multivitamin protocol includes the following: thiamine, 25 mg; riboflavin, 5 mg; niacin, 50 mg; niacinamide, 50 mg; pantothenic acid, 12.5 mg; pyridoxine, 7.5 mg; ascorbic acid, 500 mg; vitamin A, 5,000 IU; vitamin D, 500 IU; vitamin E, 2.5 IU.

Table 4. Composition of Intramuscular Protocol B

Nutrient

Concentration

Volume

Magnesium sulf.

500 mg/ml

3 ml

Calcium gly/lac

10 mg/ml

4 ml

Vitamin B12

10,000 mcg/ml

0.5 ml

Summary

The proposed integrative model of clinical allergy evolved in three phases. In the first phase, the focus was on classical immunology in general, and on in vitro methodologies for the diagnosis of IgE-mediated disorders in particular. In the second phase, the focus shifted to ecologic issues concerning the allergic individual. In the third phase, emphasis was on issues of oxidosis and dysoxygenosis that greatly amplify IgE-mediated responses. Furthermore, allergic symptomatology was seen within the broader context of oxidative-dysoxygenative injury to the bowel, blood, and liver ecosystems. A case is made for adding adjunctive nutrient, herbal, and redox-restorative therapies to the specific antigen immunotherapy for improving clinical outcome. Such adjunctive therapies were found to be especially beneficial in cases in which allergic symptomatology is associated with microscopic evidence of oxidative coagulopathy and clinical features of persistent fatigue, myalgia, abdominal symptoms, and cognitive dysfunctions.

 


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